{"id":2084,"date":"2011-03-01T00:00:00","date_gmt":"2011-03-01T08:00:00","guid":{"rendered":"https:\/\/vermont.salk.edu\/news-release\/scientists-discover-genetic-switch-that-increases-muscle-blood-supply\/"},"modified":"2011-03-01T00:00:00","modified_gmt":"2011-03-01T08:00:00","slug":"scientists-discover-genetic-switch-that-increases-muscle-blood-supply","status":"publish","type":"disclosure","link":"https:\/\/www.salk.edu\/es\/news-release\/scientists-discover-genetic-switch-that-increases-muscle-blood-supply\/","title":{"rendered":"Cient\u00edficos descubren interruptor gen\u00e9tico que aumenta el suministro de sangre a los m\u00fasculos"},"content":{"rendered":"<p>LA JOLLA, CA\u2014Muchas personas sufren de una condici\u00f3n devastadora conocida como isquemia cr\u00edtica de miembros (ICM) que puede causar atrofia muscular e incluso amputaci\u00f3n. La enfermedad est\u00e1 relacionada con el bloqueo del flujo sangu\u00edneo al m\u00fasculo esquel\u00e9tico y las opciones de tratamiento actuales incluyen ejercicio de rehabilitaci\u00f3n y bypass quir\u00fargico de los vasos sangu\u00edneos. Una nueva investigaci\u00f3n precl\u00ednica sugiere que puede haber una manera de restaurar el suministro de sangre en el m\u00fasculo esquel\u00e9tico sin intervenci\u00f3n tradicional.\n<\/p>\n<p>\nCient\u00edficos del Centro de Ciencias de la Salud de la Universidad de Texas en Houston (UTHealth) y del Instituto Salk de Estudios Biol\u00f3gicos anunciaron en la edici\u00f3n impresa del 2 de marzo de la revista <em>Metabolismo celular<\/em> que han identificado un interruptor gen\u00e9tico que puede aumentar el n\u00famero de vasos sangu\u00edneos en el m\u00fasculo esquel\u00e9tico de ratones que no hacen ejercicio.\n<\/p>\n<div class=\"imageCaption\"><img decoding=\"async\" src=\"https:\/\/www.salk.edu\/wp-content\/uploads\/2011\/01\/CMCover.jpg\" alt=\"Genetic enhancement of skeletal muscle blood supply\" width=\"300\"><\/p>\n<p>Mejora gen\u00e9tica del suministro sangu\u00edneo del m\u00fasculo esquel\u00e9tico.\n<\/p>\n<p>\nActivar un interruptor gen\u00e9tico conocido como receptor de estr\u00f3geno gamma convierte las fibras musculares de contracci\u00f3n r\u00e1pida en fibras de contracci\u00f3n lenta con un denso suministro de vasos sangu\u00edneos. La imagen compuesta muestra vasos sangu\u00edneos en verde y fibras musculares en rojo.\n<\/p>\n<p>\nImagen: Cortes\u00eda de Jamie Simon, Instituto Salk de Estudios Biol\u00f3gicos<\/p>\n<\/div>\n<p>\nEl m\u00fasculo esquel\u00e9tico est\u00e1 compuesto por dos tipos de fibras: las fibras de contracci\u00f3n lenta que intr\u00ednsecamente tienen un suministro denso de vasos sangu\u00edneos y las fibras de contracci\u00f3n r\u00e1pida que tienen menos vasos sangu\u00edneos. Los investigadores utilizaron un interruptor gen\u00e9tico conocido como receptor gamma relacionado con los estr\u00f3genos (ERR gamma) que, cuando se activa en las fibras de contracci\u00f3n r\u00e1pida de ratones mediante ingenier\u00eda gen\u00e9tica, convierte estas fibras en fibras de contracci\u00f3n lenta.\n<\/p>\n<p>\n\u201cEsto, en consecuencia, result\u00f3 en un marcado aumento del suministro de sangre a los m\u00fasculos, medido por im\u00e1genes y angiograf\u00eda\u201d, dijo Vihang Narkar, Ph.D., investigador principal y profesor asistente de medicina molecular en la Facultad de Medicina de UTHealth. \u201cEstos m\u00fasculos gen\u00e9ticamente transformados tambi\u00e9n adquieren otras caracter\u00edsticas de los m\u00fasculos de contracci\u00f3n lenta, como una mayor capacidad metab\u00f3lica y resistencia a la fatiga, que pueden ser beneficiosas adem\u00e1s para resolver enfermedades vasculares musculares.\u201d\n<\/p>\n<p>\nNarkar, cuyo laboratorio en UTHealth est\u00e1 ubicado en el Centro de Investigaci\u00f3n de Diabetes y Obesidad del Instituto Brown de Medicina Molecular para la Prevenci\u00f3n de Enfermedades Humanas, dijo: \u201cLa identificaci\u00f3n del interruptor vascular del receptor gamma relacionado con los estr\u00f3genos abrir\u00e1 v\u00edas terap\u00e9uticas potenciales para tratar la CLI y otras enfermedades cardiovasculares relacionadas con un suministro de sangre defectuoso\u201d.\u201d\n<\/p>\n<p>\nColin Barker, M.D., profesor asistente de cardiolog\u00eda en la Facultad de Medicina de UTHealth, dijo que se necesita nueva investigaci\u00f3n para ayudar a las personas con enfermedad arterial perif\u00e9rica, particularmente aquellas con la forma m\u00e1s grave: isquemia cr\u00edtica de miembros. \u201cLa mala circulaci\u00f3n en las piernas puede provocar atrofia muscular, infecciones, dolor severo y amputaci\u00f3n\u201d, dijo. \u201cEl trabajo del Dr. Narkar potencialmente tiene muchas aplicaciones \u00fatiles. Est\u00e1 muy dentro del \u00e1mbito de la medicina traslacional\u201d.\u201d\n<\/p>\n<p>\n\u201cComprender la red gen\u00e9tica que especifica un alto suministro vascular al m\u00fasculo nos brinda una herramienta nueva y muy poderosa para promover un mejor rendimiento muscular y la promesa de un estado f\u00edsico, especialmente para aquellos que no pueden hacer ejercicio\u201d, dice <a href=\"\/es\/faculty\/evans.html\/\">Ronald M. Evans<\/a>, doctor en filosof\u00eda, autor principal, investigador del Instituto M\u00e9dico Howard Hughes y profesor en el Laboratorio de Expresi\u00f3n G\u00e9nica del Instituto Salk. \u201cEsta es una buena noticia para las personas con enfermedades card\u00edacas, fragilidad, enfermedad vascular perif\u00e9rica y, en t\u00e9rminos m\u00e1s generales, para aquellos con una variedad de problemas m\u00e9dicos donde el ejercicio podr\u00eda ser beneficioso pero no es posible de realizar\u201d.\u201d\n<\/p>\n<p>\nEn 2010, se estim\u00f3 que entre 2.8 y 3.5 millones de ciudadanos estadounidenses padec\u00edan isquemia cr\u00edtica de miembros, seg\u00fan un informe de THE SAGE GROUP, una empresa independiente de investigaci\u00f3n y consultor\u00eda especializada en enfermedad arterial perif\u00e9rica. Los factores de riesgo de la CLI incluyen la diabetes, la obesidad y el tabaquismo.\n<\/p>\n<p>\n\u201cEl ejercicio es una parte importante de cualquier estrategia de intervenci\u00f3n para prevenir o tratar la diabetes mellitus y la obesidad\u201d, dijo Perry Bickel, M.D., profesor asociado de medicina en la UTHealth Medical School y director del UTHealth Center for Diabetes and Obesity Research. \u201cLos resultados de los doctores Narkar y Evans apoyan la noci\u00f3n de que en el futuro podr\u00edamos dise\u00f1ar medicamentos que produzcan los beneficios del ejercicio para contrarrestar el da\u00f1o que la diabetes y la obesidad causan al cuerpo, como los bloqueos de los vasos sangu\u00edneos\u201d.\u201d\n<\/p>\n<p>\nNarkar y Evans colaboraron en un estudio muy publicitado publicado en la revista <em>C\u00e9lula<\/em> en 2008 en el que se utilizaron dos f\u00e1rmacos mim\u00e9ticos experimentales de ejercicio: GW1516 y AICAR, para aumentar la resistencia de ratones que no realizaban ejercicio. Estos f\u00e1rmacos act\u00faan sobre diferentes interruptores gen\u00e9ticos, a saber, PPAR delta y AMPK.\n<\/p>\n<p>\nLa asistente de investigaci\u00f3n de UTHealth, Sabina Lorca, tambi\u00e9n contribuy\u00f3 al estudio. Los colaboradores del Instituto Salk incluyen a: Weiwei Fan, Ph.D.; Michael Downes, Ph.D.; Ruth T. Yu., Ph.D.; Johan W. Jonker, Ph.D.; William A. Alaynick, Ph.D.; Ester Banayo; y Malith S. Karunasiri.\n<\/p>\n<p>\nEl estudio, titulado \u201cExercise and PGC1-alpha-Independent Synchronization of Type I Muscle Metabolism and Vasculature by ERR gamma\u201d, fue financiado por donaciones del Howard Hughes Medical Institute, la Larry L. Hillblom Foundation, el Nuclear Receptor Signaling Atlas, The Leona M. and Harry B. Helmsley Charitable Trust y los National Institutes of Health (NIH) a Evans. Las subvenciones de los NIH y la Muscular Dystrophy Association a Narkar tambi\u00e9n apoyaron este estudio.\n<\/p>\n<p><strong><br \/>\nAcerca del Instituto Salk de Estudios Biol\u00f3gicos:<\/strong><br \/>\nEl Instituto Salk de Estudios Biol\u00f3gicos es una de las instituciones de investigaci\u00f3n b\u00e1sica m\u00e1s destacadas del mundo, donde un cuerpo docente de prestigio internacional investiga cuestiones fundamentales de las ciencias de la vida en un entorno \u00fanico, colaborativo y creativo. Centrados tanto en el descubrimiento como en la formaci\u00f3n de las futuras generaciones de investigadores, los cient\u00edficos del Salk realizan contribuciones revolucionarias a nuestra comprensi\u00f3n del c\u00e1ncer, el envejecimiento, el Alzheimer, la diabetes y las enfermedades infecciosas mediante el estudio de la neurociencia, la gen\u00e9tica, la biolog\u00eda celular y vegetal, y otras disciplinas relacionadas.\n<\/p>\n<p>\nLos logros del cuerpo docente han sido reconocidos con numerosos galardones, entre los que se incluyen premios Nobel y la pertenencia a la Academia Nacional de Ciencias. Fundado en 1960 por el Dr. Jonas Salk, pionero en la vacuna contra la poliomielitis, el Instituto es una organizaci\u00f3n independiente sin fines de lucro y un hito arquitect\u00f3nico.\n<\/p>\n<p><strong><br \/>\nSobre The University of Texas Health Science Center at Houston (UTHealth):<br \/>\n<\/strong><br \/>\nEl Centro de Ciencias de la Salud de la Universidad de Texas en Houston (UTHealth), el sistema acad\u00e9mico de salud m\u00e1s completo del Sistema de la Universidad de Texas y de la regi\u00f3n de la Costa del Golfo de los EE. UU., alberga escuelas de inform\u00e1tica biom\u00e9dica, ciencias biom\u00e9dicas, odontolog\u00eda, medicina, enfermer\u00eda y salud p\u00fablica. Tambi\u00e9n incluye un hospital psiqui\u00e1trico, m\u00faltiples institutos y centros, una red en crecimiento de cl\u00ednicas y programas de extensi\u00f3n en educaci\u00f3n y atenci\u00f3n en toda la regi\u00f3n. Los principales hospitales de ense\u00f1anza de la universidad incluyen el Memorial Hermann-Texas Medical Center, el Children's Memorial Hermann Hospital y el Lyndon B. Johnson General Hospital. Fundada en 1972, su profesorado, personal y estudiantes est\u00e1n comprometidos a ofrecer soluciones innovadoras que creen la mejor esperanza para un futuro m\u00e1s saludable.<\/p>","protected":false},"featured_media":0,"template":"","faculty":[91],"disease-research":[],"class_list":["post-2084","disclosure","type-disclosure","status-publish","hentry","faculty-ronald-evans"],"acf":[],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v27.3 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Scientists discover genetic switch that increases muscle blood supply - Salk Institute for Biological Studies<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/www.salk.edu\/es\/news-release\/scientists-discover-genetic-switch-that-increases-muscle-blood-supply\/\" \/>\n<meta property=\"og:locale\" content=\"es_MX\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Scientists discover genetic switch that increases muscle blood supply - Salk Institute for Biological Studies\" \/>\n<meta property=\"og:description\" content=\"LA JOLLA, CA\u2014Many people suffer from a devastating condition known as critical limb ischemia (CLI) that can lead to muscle wasting and even amputation. 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